Version 5.4
Bos taurus Protein: BT.35142
Summary
InnateDB Protein IDBP-690868.2
Last Modified 2014-10-13 [Report errors or provide feedback]
Gene Symbol BT.35142
Protein Name deleted in bladder cancer protein 1 homolog precursor
Synonyms DBC1;
Species Bos taurus
Ensembl Protein ENSBTAP00000021192
InnateDB Gene IDBG-638770 (BT.35142)
Protein Structure
UniProt Annotation
Function
Subcellular Localization
Disease Associations
Tissue Specificity
Comments
Interactions
Number of Interactions This gene and/or its encoded proteins are associated with 0 experimentally validated interaction(s) in this database.
They are also associated with 7 interaction(s) predicted by orthology.
Predicted by orthology
Total 7 [view]
Gene Ontology

Molecular Function
Accession GO Term
GO:0005515 protein binding
Biological Process
GO:0008219 cell death
GO:0045666 positive regulation of neuron differentiation
GO:0045786 negative regulation of cell cycle
GO:0045930 negative regulation of mitotic cell cycle
GO:0071300 cellular response to retinoic acid
Cellular Component
GO:0005737 cytoplasm
Protein Structure and Domains
PDB ID
InterPro IPR020864 Membrane attack complex component/perforin (MACPF) domain
PFAM PF01823
PRINTS
PIRSF
SMART SM00457
TIGRFAMs
Post-translational Modifications
Modification
Cross-References
SwissProt
PhosphoSite PhosphoSite-
TrEMBL F1N6R0
UniProt Splice Variant
Entrez Gene 538990
UniGene Bt.35142
RefSeq NP_001015669
HUGO HGNC:2687
OMIM
CCDS
HPRD
IMGT
EMBL DAAA02024683 DAAA02024684 DAAA02024685 DAAA02024686 DAAA02024687
GenPept

If you use InnateDB for your research, please cite the following publication:

Breuer et al., InnateDB: systems biology of innate immunity and beyond - recent updates and continuing curation. Nucl. Acids Res. (2013) 41 (D1) 

InnateDB is being developed jointly by the Brinkman Laboratory (Simon Fraser University, British Columbia, Canada), the Hancock Laboratory (University of British Columbia, Vancouver, British Columbia) and the Lynn EMBL Australia Group (South Australian Health & Medical Research Institute and Flinders University, Adelaide, Australia).

Funding is currently provided by Allergen and EMBL Australia. Previous funding has been provided by Genome Canada, the Foundation for the National Institutes of Health through the Grand Challenges in Global Health initiative and by Teagasc. InnateDB curated interactions are licensed under the Design Science License. All other data is licensed under the terms of the originating database. Contact: innatedb-mail@sfu.ca