Version 5.4
Bos taurus Protein: CYP1B1
Summary
InnateDB Protein IDBP-684185.2
Last Modified 2014-10-13 [Report errors or provide feedback]
Gene Symbol CYP1B1
Protein Name Uncharacterized protein
Synonyms
Species Bos taurus
Ensembl Protein ENSBTAP00000013922
InnateDB Gene IDBG-632563 (CYP1B1)
Protein Structure
UniProt Annotation
Function
Subcellular Localization
Disease Associations
Tissue Specificity
Comments
Interactions
Number of Interactions This gene and/or its encoded proteins are associated with 0 experimentally validated interaction(s) in this database.
They are also associated with 2 interaction(s) predicted by orthology.
Predicted by orthology
Total 2 [view]
Gene Ontology

Molecular Function
Accession GO Term
GO:0004497 monooxygenase activity
GO:0005506 iron ion binding
GO:0016491 oxidoreductase activity
GO:0016705 oxidoreductase activity, acting on paired donors, with incorporation or reduction of molecular oxygen
GO:0016712 oxidoreductase activity, acting on paired donors, with incorporation or reduction of molecular oxygen, reduced flavin or flavoprotein as one donor, and incorporation of one atom of oxygen
GO:0020037 heme binding
GO:0046872 metal ion binding
Biological Process
GO:0001525 angiogenesis
GO:0002930 trabecular meshwork development
GO:0006725 cellular aromatic compound metabolic process
GO:0006805 xenobiotic metabolic process
GO:0006809 nitric oxide biosynthetic process
GO:0007155 cell adhesion
GO:0008202 steroid metabolic process
GO:0008210 estrogen metabolic process
GO:0008285 negative regulation of cell proliferation
GO:0008631 intrinsic apoptotic signaling pathway in response to oxidative stress
GO:0009404 toxin metabolic process
GO:0009636 response to toxic substance
GO:0010575 positive regulation vascular endothelial growth factor production
GO:0019369 arachidonic acid metabolic process
GO:0030199 collagen fibril organization
GO:0030336 negative regulation of cell migration
GO:0032088 negative regulation of NF-kappaB transcription factor activity
GO:0033629 negative regulation of cell adhesion mediated by integrin
GO:0042572 retinol metabolic process
GO:0042574 retinal metabolic process
GO:0043065 positive regulation of apoptotic process
GO:0043542 endothelial cell migration
GO:0045766 positive regulation of angiogenesis
GO:0046427 positive regulation of JAK-STAT cascade
GO:0046466 membrane lipid catabolic process
GO:0048514 blood vessel morphogenesis
GO:0055114 oxidation-reduction process
GO:0061298 retina vasculature development in camera-type eye
GO:0061304 retinal blood vessel morphogenesis
GO:0070301 cellular response to hydrogen peroxide
GO:0071407 cellular response to organic cyclic compound
GO:0071603 endothelial cell-cell adhesion
GO:1901313 positive regulation of gene expression involved in extracellular matrix organization
GO:2000377 regulation of reactive oxygen species metabolic process
Cellular Component
GO:0005739 mitochondrion
Protein Structure and Domains
PDB ID
InterPro IPR001128 Cytochrome P450
IPR002401 Cytochrome P450, E-class, group I
IPR002403 Cytochrome P450, E-class, group IV
PFAM PF00067
PRINTS PR00385
PR00463
PR00465
PIRSF
SMART
TIGRFAMs
Post-translational Modifications
Modification
Cross-References
SwissProt
PhosphoSite PhosphoSite-
TrEMBL F1MNG1
UniProt Splice Variant
Entrez Gene 511470
UniGene Bt.59077
RefSeq NP_001179223
HUGO
OMIM
CCDS
HPRD
IMGT
EMBL AB373013 DAAA02030581
GenPept BAF95891

If you use InnateDB for your research, please cite the following publication:

Breuer et al., InnateDB: systems biology of innate immunity and beyond - recent updates and continuing curation. Nucl. Acids Res. (2013) 41 (D1) 

InnateDB is being developed jointly by the Brinkman Laboratory (Simon Fraser University, British Columbia, Canada), the Hancock Laboratory (University of British Columbia, Vancouver, British Columbia) and the Lynn EMBL Australia Group (South Australian Health & Medical Research Institute and Flinders University, Adelaide, Australia).

Funding is currently provided by Allergen and EMBL Australia. Previous funding has been provided by Genome Canada, the Foundation for the National Institutes of Health through the Grand Challenges in Global Health initiative and by Teagasc. InnateDB curated interactions are licensed under the Design Science License. All other data is licensed under the terms of the originating database. Contact: innatedb-mail@sfu.ca