Version 5.4
Bos taurus Protein: EDAR
Summary
InnateDB Protein IDBP-686148.2
Last Modified 2014-10-13 [Report errors or provide feedback]
Gene Symbol EDAR
Protein Name ectodysplasin A receptor
Synonyms
Species Bos taurus
Ensembl Protein ENSBTAP00000026148
InnateDB Gene IDBG-634358 (EDAR)
Protein Structure
UniProt Annotation
Function
Subcellular Localization
Disease Associations
Tissue Specificity
Comments
Interactions
Number of Interactions This gene and/or its encoded proteins are associated with 0 experimentally validated interaction(s) in this database.
They are also associated with 7 interaction(s) predicted by orthology.
Predicted by orthology
Total 7 [view]
Gene Ontology

Molecular Function
Accession GO Term
GO:0004872 receptor activity
GO:0005515 protein binding
Biological Process
GO:0001942 hair follicle development
GO:0007165 signal transduction
GO:0042346 positive regulation of NF-kappaB import into nucleus
GO:0042475 odontogenesis of dentin-containing tooth
GO:0043473 pigmentation
GO:0060662 salivary gland cavitation
Cellular Component
GO:0005886 plasma membrane
GO:0045177 apical part of cell
Protein Structure and Domains
PDB ID
InterPro IPR000488 Death domain
IPR011029 Death-like domain
PFAM PF00531
PRINTS
PIRSF
SMART SM00005
TIGRFAMs
Post-translational Modifications
Modification
Cross-References
SwissProt
PhosphoSite PhosphoSite-
TrEMBL E1BBS7
UniProt Splice Variant
Entrez Gene 613947
UniGene
RefSeq XP_002691323
HUGO HGNC:2895
OMIM
CCDS
HPRD
IMGT
EMBL DAAA02031033 DAAA02031034
GenPept

If you use InnateDB for your research, please cite the following publication:

Breuer et al., InnateDB: systems biology of innate immunity and beyond - recent updates and continuing curation. Nucl. Acids Res. (2013) 41 (D1) 

InnateDB is being developed jointly by the Brinkman Laboratory (Simon Fraser University, British Columbia, Canada), the Hancock Laboratory (University of British Columbia, Vancouver, British Columbia) and the Lynn EMBL Australia Group (South Australian Health & Medical Research Institute and Flinders University, Adelaide, Australia).

Funding is currently provided by Allergen and EMBL Australia. Previous funding has been provided by Genome Canada, the Foundation for the National Institutes of Health through the Grand Challenges in Global Health initiative and by Teagasc. InnateDB curated interactions are licensed under the Design Science License. All other data is licensed under the terms of the originating database. Contact: innatedb-mail@sfu.ca