Version 5.4
Bos taurus Gene: CLEC11A
Summary
InnateDB Gene IDBG-641217.3
Last Modified 2014-10-13 [Report errors or provide feedback]
Gene Symbol CLEC11A
Gene Name C-type lectin domain family 11 member A precursor
Synonyms
Species Bos taurus
Ensembl Gene ENSBTAG00000005576
Encoded Proteins
IDBP-693550
C-type lectin domain family 11 member A
Protein Structure
Useful resources Stemformatics EHFPI ImmGen
Entrez Gene
Summary This gene does not have any Entrez summary - the following is the summary from its human ortholog ENSG00000105472:
This gene encodes a member of the C-type lectin superfamily. The encoded protein is a secreted sulfated glycoprotein and functions as a growth factor for primitive hematopoietic progenitor cells. An alternative splice variant has been described but its biological nature has not been determined. [provided by RefSeq, Jul 2008]
Gene Information
Type Protein coding
Genomic Location Chromosome 18:57259806-57262108
Strand Forward strand
Band
Transcripts
ENSBTAT00000007326 ENSBTAP00000007326
Interactions
Number of Interactions This gene and/or its encoded proteins are associated with 0 experimentally validated interaction(s) in this database.
Gene Ontology

Molecular Function
Accession GO Term
GO:0008083 growth factor activity
GO:0030246 carbohydrate binding
Biological Process
GO:0008284 positive regulation of cell proliferation
Cellular Component
GO:0005576 extracellular region
GO:0005615 extracellular space
Orthologs
Species
Homo sapiens
Mus musculus
Gene ID
Gene Order
ENSG00000105472
View Gene Order
ENSMUSG00000004473
View Gene Order
Cross-References
SwissProt
TrEMBL A5D7L1
UniProt Splice Variant
Entrez Gene 522611
UniGene Bt.16680
RefSeq NM_001098054
HUGO
OMIM
CCDS
HPRD
IMGT
EMBL BC140598 DAAA02047520
GenPept AAI40599
RNA Seq Atlas 522611

If you use InnateDB for your research, please cite the following publication:

Breuer et al., InnateDB: systems biology of innate immunity and beyond - recent updates and continuing curation. Nucl. Acids Res. (2013) 41 (D1) 

InnateDB is being developed jointly by the Brinkman Laboratory (Simon Fraser University, British Columbia, Canada), the Hancock Laboratory (University of British Columbia, Vancouver, British Columbia) and the Lynn EMBL Australia Group (South Australian Health & Medical Research Institute and Flinders University, Adelaide, Australia).

Funding is currently provided by Allergen and EMBL Australia. Previous funding has been provided by Genome Canada, the Foundation for the National Institutes of Health through the Grand Challenges in Global Health initiative and by Teagasc. InnateDB curated interactions are licensed under the Design Science License. All other data is licensed under the terms of the originating database. Contact: innatedb-mail@sfu.ca